What is a consequence of mucosal inflammation during an asthma attack?

Mucosal inflammation during an asthma attack leads to hypersecretion of mucus as a response to the underlying inflammatory processes. When the airways become inflamed, they trigger the goblet cells and mucus glands to produce excessive mucus. This can obstruct airflow and contribute to the characteristic wheezing and difficulty breathing experienced during an asthma attack.

The process of mucosal inflammation is part of the body’s immune response, which aims to protect the airways but results in an overproduction of mucus. This mucus can accumulate in the airways, further narrowing them and worsening respiratory symptoms.

In contrast to this correct answer, increased airflow is unlikely during an asthma attack, as inflammation and mucus buildup can actually impede airflow. Reduced eosinophil activity would not be a consequence of mucosal inflammation; rather, eosinophils are often involved in the inflammatory response in asthma. Additionally, thinning of the bronchioles does not occur during inflammation; in fact, inflammation typically leads to swelling and thickening of the airway walls.